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Front Pharmacol (2022) 13:927219

S.LiWangA.PartchC

Future studies could test the hypothesis that the cancer-protective benefit of ferroptosis involves the reciprocal acceleration of aging

CAFs are key mediators of this process, increasing the extracellular cysteine supply in PDAC via their transsulfuration pathway to support the synthesis of glutathione (GSH) and prevent PDAC ferroptosis, a process activated by the TGF/SMAD3/ATF4 signaling pathway [91]